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An exopolysaccharide-rich fraction from Bifidobacterium animalis subsp. lactis MG741 ameliorates high-fat diet-induced MASLD by enhancing intestinal barrier function.

  • 2026-04
  • International journal of biological macromolecules 359
    • Hee-Jin Kim
    • Hee-Kyoung Son
    • Yu Ra Lee
    • Yejin Ahn
    • Hye-Bin Lee
    • Ji-Yeon Lee
    • Miri Park
    • Jae-Ho Park
    • Ho-Young Park
Exopolysaccharides (EPSs) produced by microorganisms have attracted interest as bioactive biopolymers, yet their roles in metabolic dysfunction-associated steatotic liver disease (MASLD) remain incompletely understood. Here, we prepared an EPS-rich fraction from Bifidobacterium animalis subsp. lactis MG741 (EPS-BL) by ethanol precipitation and dialysis, characterized its major molecular features, and evaluated its prebiotic and barrier-supporting activities in vitro and in a high-fat diet (HFD)-induced MASLD mouse model. EPS-BL was a mannose-dominant carbohydrate-rich preparation (219.38 mg/g mannose; weight-average molecular weight, 58.3 kDa) that also contained substantial protein, and should therefore be interpreted as a minimally processed EPS-rich material rather than a fully purified polysaccharide. In vitro, EPS-BL promoted the growth of L. plantarum and L. rhamnosus and upregulated mucin-related genes (MUC2 and MUC5AC) in LS174T goblet cells. In vivo, oral administration of EPS-BL (5 or 20 mg/kg, 8 weeks) reduced weight gain and fat mass, improved glucose-related indices, lowered circulating endotoxin, and ameliorated colonic and hepatic abnormalities in HFD-fed mice. EPS-BL also attenuated hepatic triglyceride accumulation, accompanied by suppression of lipogenesis-related proteins. Targeted metabolomics revealed coordinated shifts in serum and fecal metabolites associated with metabolic dysfunction biomarkers. Overall, EPS-BL mitigates HFD-induced MASLD in association with reinforcement of the gut barrier and modulation of the gut-liver metabolic axis, supporting its potential as a functional prebiotic ingredient.

Research Insights

SupplementHealth OutcomeEffect TypeEffect Size
Bifidobacterium animalis subsp. lactisImproved Glucose MetabolismBeneficial
Moderate
Bifidobacterium animalis subsp. lactisImproved Gut Barrier FunctionBeneficial
Moderate
Bifidobacterium animalis subsp. lactisReduced Circulating EndotoxinBeneficial
Moderate
Bifidobacterium animalis subsp. lactisReduced Hepatic SteatosisBeneficial
Moderate
Bifidobacterium plantarumImproved Glucose MetabolismBeneficial
Moderate
Bifidobacterium plantarumImproved Gut Barrier FunctionBeneficial
Moderate
Bifidobacterium plantarumReduced Hepatic SteatosisBeneficial
Moderate
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